Showing posts with label 1993-02. Show all posts
Showing posts with label 1993-02. Show all posts

Sunday, 30 March 2014

#325: A Sea Of Troubles Engulfs Incineration

=======================Electronic Edition========================

RACHEL'S HAZARDOUS WASTE NEWS #325
---February 17, 1993---
News and resources for environmental justice.
------
Environmental Research Foundation
P.O. Box 5036, Annapolis, MD 21403
Fax (410) 263-8944; Internet: erf@igc.apc.org
==========
RACHEL-4CM = DIOXIN FOCUSED DIRECTORY
Remote Access Chemical Hazards Electronic Library.
Dioxinnz.com

=======================Original Source========================

The incineration industry has suffered a series of major setbacks in recent weeks. The federal government's showcase dioxin-burning incinerator in Jacksonville, Arkansas--to which Bill Clinton personally gave a green light one week before he was elected President (see RHWN #311)--was shut down last week by a federal judge. Lawyers for EPA (U.S. Environmental Protection Agency) admitted to U.S. District Judge Stephen Reasoner in Little Rock that the Jacksonville incinerator could not destroy 99.9999% of the dioxin fed into it, as is required by EPA regulations, whereupon the judge ordered the plant shut.

It was a stunning victory for Greenpeace chemist Pat Costner, who had been the first to reveal that incinerators could not achieve 99.9999% destruction (see RHWN #312 and #280), and for local activist-citizens like Sharon Golgan in Jacksonville who had fought the incinerator proposal for at least six years.

The nation's other showcase incinerator--built by Waste Technologies Industries (WTI) on the banks of the Ohio River in East Liverpool, Ohio--was fighting for its life in federal court in Cleveland earlier this week. Local citizens there have been opposing the WTI incinerator proposal for 12 years. EPA's Region 5 office in Chicago and Ohio state officials bent the rules, and even broke a few, to give WTI a permit to start burning wastes (see RHWN #287). The state of West Virginia, local citizens, and Greenpeace filed a lawsuit January 13 asking a federal judge to prevent WTI from conducting a "test burn."[1] As soon as a test burn is completed, the WTI incinerator can begin commercial operation for up to a year while EPA evaluates the test results. (Vice-President Al Gore said December 7, 1992, that he and Mr. Clinton would stop WTI from proceeding until a study was completed of all health issues, and all legal issues surrounding EPA's issuance of a permit [see RHWN #315]]--but it now appears that Mr. Gore has changed his mind.) Federal Judge Ann Aldrich in Cleveland issued a temporary restraining order against WTI January 15 and set February 16 as the day she would decide whether to issue a preliminary injunction, which would prevent WTI from conducting the trial burn until a full court trial could be held.[2]

As we go to press, it is not clear how this phase of the battle will come out. However, secret EPA documents that came to light during in court have raised serious health questions about every incinerator in the country, including solid waste incinerators.

Background
At issue in every instance is dioxin, which is created as an unwanted byproduct of incineration (and as a byproduct of other industrial processes, such as pulp and paper manufacture, and the manufacture of some pesticides). In April, 1991, EPA began a major "scientific reassessment" of dioxin and discovered that effects of dioxin can be observed in human cells at the levels of exposure now present in the environment. In other words, there's already sufficient dioxin in the environment to produce observable effects in humans. The Clinton/Gore administration knows this is a problem because a December, 1992, briefing document prepared by the EPA Transition Team says, "This [EPA's dioxin reassessment] is likely to be an extremely controversial document as there are new findings indicating adverse reproductive effects at existing environmental levels."

A Secret Risk Assessment and a Leaked Memo
EPA's position throughout the 1980s has been that incineration is safe, even though every incinerator is known to produce dioxin. To "prove" the safety of incinerators, EPA has used a technique called "risk assessment." A risk assessment estimates the amount of dioxin being released, estimates various pathways it might travel through the environment, and calculates the resulting exposures of humans. Finally, a risk assessment estimates the health effects resulting from the calculated exposures.

In the case of dioxin, over the years EPA's "standard" risk assessment has assumed that airborne dioxin only enters humans through their lungs. Dioxin that falls to the ground and is then incorporated into the food chain and eaten has always been ignored in EPA risk assessments.

However, the EPA's team of scientists conducting the official "reassessment" of dioxin's toxicity published a draft report last summer called, ESTIMATING EXPOSURE TO DIOXIN-LIKE COMPOUNDS in which they clearly stated that a proper risk assessment for an incinerator must include all routes of exposure for dioxin, not merely via the lungs.[3] It is well known that dioxins accumulate in the food chain, and that meat, milk and fish are the major sources of dioxin exposure for humans.[4]

When Greenpeace researcher Joe Thornton did his own risk assessment on the WTI incinerator, using the technique recommended in EPA's draft report, including dioxins in beef and milk, he found that WTI posed risks 10000 times higher than EPA had calculated. To counter Thornton, EPA did its own food-chain risk assessment, which was not released to the public, but which came to light in court.[5] The EPA's secret risk assessment concludes that dioxin from WTI is 1000 times more dangerous than the "official" published EPA risk assessment says it is.

Leaving aside the serious ethical issue of EPA refusing to publish important health and safety information about WTI, an internal memo from Richard Guimond, acting chief of EPA's Office of Solid Waste and Emergency Response, dated January 22, 1993, leaked to Greenpeace, says, "There are very serious implications associated with adopting risk assessment procedures based on indirect exposure routes for air emission sources."[6] Translation: if food-chain exposures are now to be counted in incinerator risk assessments, many incinerators will be found to be unacceptably dangerous.

The new understanding of dioxin--that it's already present in the environment at levels that affect humans--plus the inclusion of food-chain exposures in risk assessments, plus the failure of incinerators to achieve the destruction efficencies required by regulations--all seem to add up to a mushrooming debacle for the incineration industry.

What are the alternatives to incineration?

Jacksonville holds the key

EPA scientists have known since 1985 that incinerators cannot achieve 99.9999% efficiency in destroying wastes present in low concentrations (see RHWN #280 and #312a.), but EPA officials have stated at hundreds of public presentations since 1985 that 99.9999% could be achieved. Lying to the public carries no penalty, but lying to a judge is a different matter. At the hearing in Little Rock February 12, EPA was represented by U.S. Justice Department lawyer Ron Spritzer. Judge Reasoner said to Spritzer, "Indulge me for a moment. If I asked you to prove that you could achieve a six 9 DRE [99.9999% destruction and removal efficiency] on dioxin, could you physically produce technological data that shows that?" "No sir, we could not," said Mr. Spritzer.[7]

That damaging admission was sufficient for the judge. He ordered the plant shut. EPA is considering appealing the judge's verdict, but they do not appear to have a strong case. In all likelihood, the Jacksonville incinerator is shut for good.

The Jacksonville incinerator was in considerable trouble even before Judge Reasoner's decision. On January 22, an Arkansas state official revealed that the incinerator was producing a larger volume of hazardous waste than it was destroying. The machine had been set up in a residential neighborhood of Jacksonville to "destroy" 30,000 drums of hazardous liquids left over from a defunct chemical factory that had made chemical warfare agents on the site for many years (most recently, herbicides for Vietnam). Local citizens had recommended moving the waste out of town by rail, or simply building a concrete mausoleum on the site to contain the waste safely until someone found a way to detoxify it. But Arkansas and federal EPA officials insisted that incineration was the safest, cheapest solution to the problem. They convinced then-Governor Bill Clinton to put up $10.7 million of state money to build and operate the incinerator.

At the end of a year's burning, 9,600 drums of waste had been "destroyed" by the Jacksonville incinerator, but in the process the incinerator had created 12,000 drums of salt and another 1730 drums of ash (13,730 drums total) for a net gain of 43% in the volume of waste. Furthermore, the salt and the ash are so laced with dioxin that they are legally a "hazardous waste" and thus cannot be taken off the site.

"We did not anticipate this," said Doug Szenher, a spokesperson for the Arkansas state department of Pollution Control and Ecology. "The whole idea was that the salt and ash was to have been de-listed [declared non-hazardous] and taken to a landfill site. It just didn't work out that way," he said.[8]


So after spending $7 million burning chemicals fruitlessly for a year, EPA is now spending $400,000 to build a 30,000-square-foot building on the site, to store the hazardous salt and ash until someone can figure out how to detoxify it. So far as we know, this is the first above-ground mausoleum built to store hazardous waste. (See RHWN #260.) In Jacksonville, above-ground concrete storage will soon be a real, demonstrated alternative to incineration.


--Peter Montague, Ph.D.

===============

[1] GREENWIRE staff, "WTI Incinerator: Foes File Suit in Effort to Block Test," GREENWIRE Vol. 2 No. 171 (January 14, [1993).] GREENWIRE is "the daily executive briefing on the environment" available on-line; phone (703) 237-5130.

[2] GREENWIRE staff, "Incinerator: WTI Counters with Suit of its Own," GREENWIRE Vol. 2 No. 172 (January 15, 1993). GREENWIRE is "the daily executive briefing on the environment" available on-line; phone (703) 237-5130.

[3] U.S. Environmental Protection Agency, ESTIMATING EXPOSURE TO DIOXIN-LIKE COMPOUNDS [EPA/600/6-88/005B] WORKSHOP REVIEW DRAFT (Washington, D.C.: U.S. Environmental Protection Agency, August, 1992).

[4] U.S. Environmental Protection Agency, cited above, Appendix B.

[5] Memo from William Farland, director of EPA's Office of Health and Environmental Assessment, to Brian Grant, U.S. Department of Justice, "WTI Screening Level Analysis," dated Feb. 8, 1993, attaching a 21-page risk assessment called "Screening Level Analysis of Impacts From WTI Facility," dated Feb. 5, 1993.

[6] Memo from Richard Guimond, Acting Assistant Administrator, Office of Solid Waste and Emergency response, to EPA Administrator Carol Browner, "WTI Incinerator Issues," dated January 22, 1993. 2 pgs.

[7] Sandy Davis, "Judge Halts Dioxin Burn at Vertac," ARKANSAS DEMOCRAT GAZETTE February 13, 1993, pgs. 1A, 13A.

[8] Sandy Davis, "At vertac, more waste rolls out than in," ARKANSAS DEMOCRAT GAZETTE January 24, 1993, pg. 12A.

Descriptor terms: incineration; epa; oh; risk assessment; ar; dioxin; wti; waste technologies inc; hazardous wastes; health; jacksonville; east liverpool; waste disposal technologies; waste treatment technologies;

#323: New Perspectives on Toxics--Part 2: Male Reproductive System Is Harmed By Toxic Exposures, Causing Birth Defects, Sterility

=======================Electronic Edition========================

RACHEL'S HAZARDOUS WASTE NEWS #323
---February 3, 1993---
News and resources for environmental justice.
------
Environmental Research Foundation
P.O. Box 5036, Annapolis, MD 21403
Fax (410) 263-8944; Internet: erf@igc.apc.org
==========
RACHEL-4CM = DIOXIN FOCUSED DIRECTORY
Remote Access Chemical Hazards Electronic Library.
Dioxinnz.com

=======================Original Source========================


The ancient Greeks observed that men heavily exposed to lead became sterile. But this knowledge was not passed to the Romans, who stored their drinking water and their wine in lead-lined containers. Romans also added lead to some of their drinks as a sweetener. Based on examination of lead in Roman bones, some historians conclude that the Roman upper classes probably couldn't reproduce themselves, contributing to the fall of Rome.[1]

Until very recently, scientists paid little attention to the effects of environmental agents on human reproduction. The modern period began in 1941 when blindness, deafness and death were reported among the offspring of pregnant women exposed to rubella (German measles). The thalidomide catastrophe in 1954-1961 brought home the potential dangers of chemicals, in this case a prescription drug. (See RHWN #322.) The birth of nearly 20,000 defective children following a Rubella epidemic in the early 1960s confirmed the association of environmental factors and birth defects.[2]

As recently as the early 1970s, few state governments were maintaining records of birth defects. In 1974 the federal government established the first national register of birth defects, monitoring hospital records that account for about 15% of all births.[3] Even today this program monitors only birth defects observed in newborns, which probably represent only about a sixth of the total defects that actually occur because many defects do not become apparent for several years.[2]

As of 1980, approximately 200,000 birth defects were estimated to have occurred in the U.S., accounting for about 7 percent of all live births. In addition, more than 560,000 infant deaths, spontaneous abortions, stillbirths and miscarriages were recorded due to defective fetal development.[2]

In 1990, the federal Centers for Disease Control (CDC) reported trends in birth defects between 1979 and 1987 in the U.S. They looked at 38 specific defects and found that 29 of the 38 had increased, two had decreased and 7 had remained stable.[3] The largest increases (29 percent and 20.2 percent) occurred in defects of the heart; no doubt some of this increase is due to better detection methods. However, there was also a 9.6 percent increase in eye defects and a 2.7 percent increase in cleft lip, so it seems likely that real increases are occurring.

As we reported last week,#322 the traditional view of birth defects highlights the role of women and disregards the role of men, even when there is good evidence showing men exposed to toxic chemicals father defective children. The traditional reason for ignoring such research is that, until recently, there has been no satisfactory theory to explain how male exposures could affect offspring, so a cause-and-effect relationship could not be established. The argument was that women are born with all the eggs they will ever have, so each egg can be exposed to toxins over a long period. Men on the other hand, produce new sperm constantly, so any individual sperm has only a brief opportunity to be exposed to toxins.

Another reason for ignoring the effect of toxins on male reproduction was the "macho sperm" theory, which said that only the fittest sperm were hardy enough to go the distance necessary to fertilize an egg.

[4] According to this theory, defective sperm could never reach an egg to fertilize it, so men couldn't be responsible for producing defective children. Now research has shown that the female reproductive tract has ways of moving sperms along whether they are healthy or defective.

Researchers used to believe that there was an effective barrier between blood vessels and the tissues where sperm originates in the testes. It is now known that the barrier is not effective against many chemicals.

Then of course there's a cultural bias, reaching back to the Salem witch trials, to blame women for trouble. "You don't have to be Sigmund Freud to figure out that there are cultural factors to say why we have paid so much attention to the female and so little to the male," says Dr. Devra Lee Davis, an epidemiologist with the National Research Council.

Research during the past decade has shown that there are two basic ways that chemicals can affect male reproduction.[5,6] Chemicals can directly affect the testes, where sperm originates. The numbers of sperm can be diminished, or some sperm can be damaged, or sperm may even carry toxins directly into the egg. Alternatively, toxins can attack the male nervous system, or endocrine system, affecting the flow of hormones that act as messengers regulating the complex chemical processes that must all work well for conception to occur.

No matter what the mechanism of damage may be, there is a growing body of evidence showing that male exposure to toxins can produce defective children. Here is a sampling:
** A nationwide study of 99,186 pregnancies in Finland showed an increased likelihood of spontaneous abortion if the father was occupationally exposed to rubber chemicals, solvents used in the manufacture of rubber products, solvents used in oil refineries, or ethylene oxide.[7]** A study of 22,192 children born with birth defects in British Columbia showed that paternal occupation as a fire fighter was related to the occurrence of heart defects. Fire fighters are often heavily exposed to carbon monoxide and to polycyclic aromatic hydrocarbons (PAHs)--the nasty chemicals in smoke and soot.[8]** A study of paternal occupation among 149 patients with Wilm's tumor (a childhood cancer of the kidney) showed that a significantly greater number of the fathers were exposed to lead on the job, compared to fathers of a control group of children without the disease.[9]** A study of 6000 men in Finland showed that paternal exposure to organic solvents nearly tripled the likelihood of spontaneous abortion as a pregnancy outcome, compared to controls not exposed to organic solvents. Painters, wood workers (for example, carpenters in the construction, furniture industry and the boat industry) were found to be at risk. The solvent toluene stood out as a particularly bad actor in his study. [10]** A study of anesthetists in the West Midlands region of England (half men, half women) showed that, during a 20-year period, 9.3% of their children were born with defects, and 31% of the anesthetists reported having trouble begetting children. Furthermore nearly all the children were born underweight. The gender of the anesthetists did not affect the likelihood of problems in their children, but female children seems to suffer greater birth defects. [11]** A recent review of several studies of paternal occupational exposures in relation to childhood cancer in the offspring showed consistently that work in hydrocarbon-related occupations (the petroleum and chemical industries), especially exposure to paint, is associated with brain cancer. Male exposure to paint is also linked to leukemias in offspring. [12]

What does all this evidence mean? It means neither men NOR women can be safely exposed to toxic chemicals. Instead of banning women from the workplace, the workplace should be cleaned up.


--Peter Montague, Ph.D.

===============

[1] S.C. Gilfillan, "Lead Poisoning and the Fall of Rome," JOURNAL OF OCCUPATIONAL MEDICINE Vol. 7 (Feb. 1965), pgs. 53-[60.]60.

[2] Raymond D. Harbison, "Teratogens," in John Doull and others, editors, CASARETT AND DOULL'S TOXICOLOGY 2nd edition (N.Y.: Macmillan, 1980), pgs. 158-175.

[3] Larry D. Edmonds and Levy M. James, "Temporal Trends in the Prevalence of Congenital Malformations at Birth Based on the Birth defects Monitoring Program, United States, [1979-1987."] MORBIDITY AND MORTALITY WEEKLY REPORTS CDC SURVEILLANCE SUMMARIES Vol. 39, No. SS-4 (December, 1990), pgs. 19-23.

[4] Sandra Blakeslee, "Research on Birth Defects Turns to Flaws in Sperm," NEW YORK TIMES (January 1, 1991), pgs. 1, 36.

[5] Lowell Sever and Nancy A. Hessol, "Toxic Effects of Occupational and Environmental Chemicals on the Testes," in J.A. Thomas and others, editors, ENDOCRINE TOXICOLOGY (N.Y.: Raven Press, 1985), pgs. 211-248.

[6] Harold Zenick, "Mechanisms of Environmental Agents by Class Associated With Adverse Male Reproductive Outcomes," in REPRODUCTION: THE NEW FRONTIER IN OCCUPATIONAL AND ENVIRONMENTAL HEALTH RESEARCH (N.Y.: Alan R. Liss, Inc., [1984),] pgs. 335-361.

[7] Marja-Lusa Lindbohm and others, "Effects of Paternal Occupational Exposure in Spontaneous Abortions," AMERICAN JOURNAL OF PUBLIC HEALTH (August, 1991), pgs. 1029-1033.

[8] Andrew F. Olshan and others, "Birth Defects Among Offspring of Firemen," AMERICAN JOURNAL OF EPIDEMIOLOGY Vol.
[131] (1990), pgs. 312-321.

[9] Arlene F. Kantor and others, "Occupations of fathers of patients with Wilm's tumour," JOURNAL OF EPIDEMIOLOGY AND COMMUNITY HEALTH Vol. 33 (1979), pgs. 253-256.

[10] Helena Taskinen and others, "Spontaneous abortions and congenital malformations among the wives of men occupationally exposed to organic solvents," SCANDINAVIAN JOURNAL OF WORK, ENVIRONMENT AND HEALTH Vol. 15 (1989), pgs. 345-352.

[11] P. J. Tomlin, "Health problems of anesthetists and their families in the West Midlands," BRITISH MEDICAL JOURNAL (24 March, 1979), pgs. 779-784.

[12] David A. Savitz and Jianjua Chen, "Parental Occupation and Childhood Cancer: Review of Epidemiological Studies," ENVIRONMENTAL HEALTH PERSPECTIVES Vol. 88 (1990), pgs. 325-337.

Descriptor terms: reproductive hazards; health; birth defects; lead; rubella; endocrine disruptors; solvents; pahs; occupational safety and health; men; males; fathers; children; spontaneous abortion; petroleum industry;