Showing posts with label 1991-09. Show all posts
Showing posts with label 1991-09. Show all posts

Tuesday, 1 April 2014

#252: Some Good News: We Could Give Up Oil

=======================Electronic Edition========================

RACHEL'S HAZARDOUS WASTE NEWS #252
---September 25, 1991---
News and resources for environmental justice.
------
Environmental Research Foundation
P.O. Box 5036, Annapolis, MD 21403
Fax (410) 263-8944; Internet: erf@igc.apc.org
==========
RACHEL-4CM = DIOXIN FOCUSED DIRECTORY
Remote Access Chemical Hazards Electronic Library.
Dioxinnz.com

========================Original Source========================

Our addiction to fossil fuels (oil and coal) is killing us. Oil alone is responsible for smog, nitrogen oxides and fine particles--in sum, urban air pollution--which is killing an estimated 30,000 to 60,000 Americans each year.[1] To these deaths we must add the regional and global consequences of oil and coal-related pollution: regional destruction of forests, crops and fish by acid rain, and the mounting threat of severe floods, harsh droughts, devastating storms, large forest fires, and ruinous declines in crop yield--which are the most likely consequences of global warming.[2] Other, lesser, consequences of our oil addiction include massive oil spills, wildlands devastation by oil exploration activities, and occasional small wars.

Now technical developments during the past eight years have made it realistic to say we can end our use of fossil fuels in the U.S. and worldwide. All we have to do is decide to do it.

A realistic alternative to oil and coal is hydrogen fuel produced by solar energy, according to a thorough study by the Center for Energy and Environmental Studies at Princeton University, funded by the National Science Foundation.[3]

According to the study, hydrogen fuel produced from water by the action of electricity--particularly electricity created by the sun's rays striking photovoltaic cells--offers a realistic substitute for all our current fossil-fuel uses high-temperature heat in industry, low-temperature heat for space-heating, and liquid fuels for transportation. Hydrogen could do it all, the study's authors say, and solar cells are the best way to make the hydrogen.

For many years, photovoltaic cells ("solar cells") have been expensive toys. A photovoltaic cell is a sheet of dark blue glass with a pair of wires attached. When the cell is placed in direct sunlight, electricity becomes available in the wires. There are no moving parts except electrons moving inside the cell, producing the electric current. (You can buy solar cells for as little as $4.00 from Edmund Scientific, 101 East Gloucester Pike, Barrington, NJ 08007-1380; phone (609) 547-8880.)

Technical advancements in the past 15 years--and especially in the last eight years--have reduced the manufacturing costs for solar cells steadily, and now such cells offer a realistic alternative to nuclear power and to coal-burning power plants. Furthermore, data from a decade of manufacturing now allows conservative estimates of continued reductions in manufacturing costs that are very likely to achieved in the next five years. The age of solar hydrogen is at hand.

As a fuel, hydrogen is almost an environmental ist's dream come true. When hydrogen burns, it emits no carbon monoxide, no carbon dioxide (the main source of global warming), no volatile organic compounds (the main source of urban smog), no fine particles (the chief killers in urban air), and no sulfur oxides (the main source of acid rain). The only pollutant created is nitrogen oxides, which can be controlled by various means (depending on the kind of combustion device the hydrogen is fueling). The main byproduct of hydrogen combustion is water vapor when the hydrogen (H) and oxygen (O) combine into H2O.

The particular solar cell technology used as the basis for the Princeton study is called amorphous silicon. Unlike older solar calls which were usually round and an inch or two in diameter, amorphous silicon cells are now manufactured by spraying a thin film onto sheets of regular plate glass 4' x 4' or even larger. The main raw material in such cells is silicon derived from sand, so no raw material shortages stand in the way of large-scale production.

Nuclear power stations and coal-fired power plants make best economic sense when they are built large (1000 megaWatts [one gigaWatt] is the average power plant today); each such plant requires an investment of billions of dollars. In contrast, there are no economies of scale to be achieved in solar-hydrogen plants beyond a 5-to-10 megaWatt facility that would cost only $4 to $10 million to manufacture. These modular units could be combined to give power output of any desired size to meet any foreseeable need. Furthermore, the modular nature of solar-hydrogen plants makes them ideal for construction of demonstration-scale units to show that real alternatives to oil are within reach.

To achieve the necessary efficiencies to make solar-hydrogen plants affordable as a total substitute for oil, they would have to be sited where the sun shines most; the world's deserts would be the best candidates for location of such facilities. The hydrogen would then be piped to consumers through gas pipelines. The land area needed to produce hydrogen equivalent to all U.S. oil would be 24,000 square miles--about 0.5% of total U.S. land area, or 7% of U.S. deserts. The land area needed to collect solar energy to produce hydrogen equivalent to the world's entire oil production in 1987 would total 205,000 square miles or about 2% of the world's desert area. ..TEXT: Photovoltaic panels need not block out the sun and kill desert vegetation; long rectangular panels on stilts, spaced appropriately to allow sunlight to strike the desert beneath them as the sun moved across the sky would allow vegetation, wildlife, and even domestic animals to thrive beneath them.

To replace all U.S. oil used today, the water needed to produce the hydrogen would add about 2% to U.S. per capita water usage. The water would not be destroyed, of course, but it would be moved from the place where the hydrogen was made to the place where the hydrogen was burned.

The Princeton study considers each of the competing alternatives and concludes that hydrogen is the best fuel and that solar cells are the best way to make the hydrogen. The study considers biomass (growing vegetation and burning it for fuel), nuclear energy, natural gas (methane), and synthetic fuels from coal. Each of these competing technologies has benefits and costs. Biomass requires 10 times as much land as solar cells and a great deal more water. Nuclear power--even if the nuclear waste problem could be solved--suffers from one unsolvable: each year a single reactor creates enough plutonium to manufacture 20 nuclear bombs, and we have seen in recent days that even facilities being actively inspected by the international atomic police can extract plutonium from reactors right beneath the policemen's nose.[4] Coal-based fuels produce carbon dioxide that promises without doubt to heat up the planet sooner or later, causing major disruptions of atmosphere and related systems (such as rain and food production). Natural gas suffers from the same unsolvable problem. ..TEXT: The German automobile firm, Daimler-Benz has produced a hydrogen-powered automobile already. The Billings Energy Corp. of Provo, Utah has manufactured a hydrogen-powered bus. Several countries (Italy, New Zealand and Canada) have already demonstrated the feasibility of a gaseous-fuel infrastructure for transportation (in other words, pipelines, tanks, filling stations, and so forth).

People fear hydrogen, principally because of the Hindenburg airship disaster at Lakehurst, NJ, in 1937, which killed 36 people. However, analysis of natural gas, gasoline and hydrogen reveals that each fuel has particular hazards associated with its manufacture, storage, transportation and use and that for each fuel procedures and precautions can be developed for safe handling.[5]

The final chapter of the Princeton study offers a scenario for shifting from an oil-based economy to a solar-hydrogen economy. Even a speedy transition would take several decades; the pace of the transition will depend upon our willingness to make the necessary investment in new facilities. Our willingness to invest depends upon our concern for the wellbeing of the planet and the services it provides to humankind.

--Peter Montague, Ph.D.
===============

 [1] The higher estimate is from recent unpublished work by U.S. Environmental Protection Agency scientist Joel Schwartz reported in the WASHINGTON POST May 13, 1991, pg. A13.

[2] These effects are the ones we should expect as the buildup of so-called greenhouse gases continues, according to Stephen Schneider of the National Center for Atmospheric Research (a federal research laboratory); see chapter 1 in Schneider's book GLOBAL WARMING (San Francisco: Sierra Club Books, 1989). ..TEXT:

[3] Joan M. Ogden and Robert H. Williams, SOLAR HYDROGEN: MOVING BEYOND FOSSIL FUELS (Washington, DC: World Resources Institute, 1989). 123 pgs.; $12.50 plus $3 shipping from WRI Publications, P.O. Box 4852 Hampden Station, Baltimore, MD 21211. Phone (800) 822-0504 or (301) 338-6963; Visa, Mastercard and purchase orders accepted.

[4] "Atomic Regulators Seek Safeguards Against Bombs," NEW YORK TIMES Sept. 24, 1991, pg. A17.

[5] The safety of hydrogen is considered in detail in J. Hord, "Is Hydrogen a Safe Fuel?" INTERNATIONAL JOURNAL OF HYDROGEN ENERGY Vol. 3 (1978), pgs. 157-176.

Descriptor terms: fossil fuels; oil; coal; energy; hydrogen fuel; solar energy; nuclear power; daimler-benz; automobiles; transportation; billings energy corp; renewable energy sources; global environmental problems; petroleum industry;

Saturday, 29 March 2014

#249 Dioxin Dangers--What's Going On?

=======================Electronic Edition========================

RACHEL'S HAZARDOUS WASTE NEWS #249
---September 4, 1991---
News and resources for environmental justice.
------
Environmental Research Foundation
P.O. Box 5036, Annapolis, MD 21403
Fax (410) 263-8944; Internet: erf@igc.apc.org
==========
RACHEL-4CM = DIOXIN FOCUSED DIRECTORY
Remote Access Chemical Hazards Electronic Library.
Dioxinnz.com
========================Original Source========================

We urge our readers to attend the First Citizens' Conference on Dioxin September 21-22, 1991, at the Omni-Europa Hotel in Chapel Hill, North Carolina. For more information phone Ellen and Paul Connett in Canton, NY: (315) 379-9200. The conference will discuss the latest scientific information on dioxin, fraud and manipulation in dioxin studies, and solutions to the dioxin problem offered by citizens.

The recent flap in the press over dioxin can all be traced back to a single government scientist who announced in April that he believes dioxin is less able to cause cancer in humans, and is therefore less dangerous, than he believed 10 years ago. In late April, Vernon N. Houk, a 62-year-old government worker, was keynote speaker at a University of Missouri conference sponsored by Syntex, a company that was being sued in a Missouri court by the family of a truck driver who had died of cancer and who had worked for years in a truck terminal that was contaminated with dioxin. Syntex faces another 350 dioxin lawsuits from individuals who lived at Times Beach, Missouri--a town the U.S. government bought out when they found it was heavily contaminated with dioxin, 10 years ago. During his speech in April, Dr. Houk dropped a bombshell: he said he believed he himself had made a mistake 10 years ago when he urged the federal government to evacuate 2200 residents from Times Beach. In August, the NEW YORK TIMES turned Dr. Houk's reassessment into two front-page stories and an editorial, the main message of which was, "Many scientists now believe dioxin isn't as bad as we thought." At least 26 other newspapers in the U.S. and Canada jumped on the bandwagon.

As the summer wore on, Dr. Houk himself went even further. In August he was quoted in the SEATTLE (Washington) TIMES (August 18, pg. B3) saying he believed the pulp and paper industry in the northwestern U.S. "has reduced dioxin levels enough to protect public health." He said this was his personal view, not a government opinion. As we saw last week,#248 the paper industry is facing so many dioxin lawsuits that it is now being compared to the asbestos industry. In a story devoted to Dr. Houk himself, the NEW YORK TIMES noted that he is now being praised and acclaimed (the TIMES said "lionized") by industrial companies that produce dioxin as a waste product, because they say he has put an end to the "dioxin scare."

Dr. Houk's views on the dangers of dioxin reflect the narrow public health perspectives of the federal government. Standards for exposure to a chemical are generally based on the chemical's ability to cause cancer, and not on its ability to cause other health problems. On this basis, Dr. Houk justifies his change of heart: "If [dioxin's] a carcinogen, it's a very weak carcinogen and Federal policy needs to reflect that," Dr. Houk says.

In actual fact there is much new scientific evidence indicating that dioxin is more dangerous than anyone knew 10 years ago. Recent evidence (summarized in a long article in the NEW YORK TIMES May 15, 1991 [pg. C4], and in CHEMICAL & ENGINEERING NEWS August 12, 1991 [pgs. 7-14]) reveals that dioxin has many toxic effects on wildlife and humans besides its ability to cause cancer. In one species of animal or another, dioxin causes wasting syndrome [progressive weight loss leading to death]; atrophy of the thymus (a blood-forming organ important in the immune system); atrophy of the spleen (another blood-forming organ important in the immune system); atrophy of the testicles; enlargement, deterioration and death of liver tissue; hyperplasia (excessive cell growth) in the urinary tract and bile ducts; birth defects; and suppression of the immune system. In addition, its ability to cause cancer in laboratory animals, wildlife and humans is now a matter of record (see RHWN #219.)

The present understanding of dioxin is that it functions like a steroid hormone. Steroid hormones are powerful chemicals that enter cells, bind to a "receptor" (a protein), form a "complex" that then attaches to the cell's chromosomes where it directly encounters the genetic material, turning on and off chemical switches that may then affect distant parts of the body in various important ways. It is not unusual for very small amounts of a steroid hormone to have major effects on the body.

In animal studies, dioxin is still the most potent poison ever seen. For example, it kills hamsters at one 64-thousandth of the fatal dose of sodium cyanide. The hamsters die of "wasting syndrome"--they lose weight and die. In rainbow trout, northern pike, and salmon, one-tenth of a part per trillion of dioxin in water causes growth retardation of young fish. The U.S. Fish and Wildlife Service calls dioxin "the most toxic synthetic compound ever tested under laboratory conditions."[1] No new evidence has changed any of this. Back when Dr. Houk was recommending that people be evacuated from Times Beach, in 1981, the danger did not seem remote or theoretical. Seventy-five horses in the town had died. Dogs, rodents, chickens, cats and birds had died. No new evidence has been found in the past 10 years to change anyone's estimate of dioxin's toxicity to these species.

In rhesus monkeys, as little as 25 parts per trillion in the diet causes increased passivity and measurable reductions in the ability to learn.[2]

"With laboratory animals, it seemed as if dioxin caused just about any effect you can think of," says Dr. Steven Safe, a toxicologist at Texas A&M University. "You name it, it did it, and at extremely low doses," he says. Dr. Safe says the mystery of dioxin has largely been solved by confirmation that TCDD [dioxin] and other toxic agents interact directly with genetic material [the chromosomes that, taken together, comprise the DNA molecule], the way hormones do. It is not unusual for a hormone to have different effects and different potencies in different species of animals. This does not make dioxin any less toxic--it merely makes it better-understood.

CHEMICAL & ENGINEERING NEWS says, "One of the most significant realizations of the past few years is that TCDD [dioxin] cannot be considered by itself... that there are a number of dioxin-like compounds that can all have toxic effects." Dioxins, dibenzofurans, PCBs, and some halogenated naphthalenes all mimic hormones; therefore scientists now believe they can all cause the range of toxic effects attributed to dioxin. This is not good news for anyone because dioxin alone may account for 7 ppt [parts per trillion] in the blood of average Americans but when these related compounds are taken into consideration, the average American may be carrying 100 ppt of dioxin equivalents in his or her blood stream. The meaning of these levels of contamination will become clearer in the next few years.

Even now, far-reaching effects in fish and birds in the Great Lakes are observable at 35 to 65 ppt. Philip M. Cook at the U.S. Environmental Protection Agency laboratory in Duluth, MN, says he believes Great Lakes fish are failing to reproduce already because of the dioxin burden they carry.

Researchers observing wildlife in the Great Lakes are seeing hermaphroditic offspring of birds--for example, male birds with partially developed female sex organs. They are seeing female-female bonding behavior. These potent dioxin-family toxins seem to act like, or interfere with, normal sex hormones like estrogen, producing "chemically castrated" males, and sexually-confused females.

Linda S. Birnbaum, director of the Environmental Toxicology Division of the EPA's Environmental Health Effects Research Laboratory has overall responsibility for the EPA's year-long reassessment of dioxin's toxicity (see RHWN #248). Unlike Vernon Houk, for whom "the issue is mostly decided" already (says CHEMICAL & ENGINEERING NEWS), Dr. Birnbaum believes when all the evidence has been evaulated, EPA may not change its regulatory number for dioxin much. The number was initially established because of a great fear of dioxin as a carcinogen. Now a whole new set of toxic properties of dioxin, and dioxin-like chemicals, have become apparent, even as the carcinogenicity question has assumed less importance.

--Peter Montague, Ph.D.

===============

[1] Ronard Eisler, DIOXIN HAZARDS TO FISH, WILDLIFE, AND INVERTEBRATES: A SYNOPTIC REVIEW. [BIOLOGICAL REPORT 85 (1.8); CONTAMINANT HAZARD REVIEWS REPORT NO. 8]. Laurel, Maryland: U.S. Department of the Interior, Fish and Wildlife Service, Patuxent Wildlife Research Center, May, 1986.

[2] R. Bowman and others. "Behavioral Effects in Monkeys Exposed to 2,3,7,8-TCDD Transmitted Maternally During Gestation and for Four Months of Nursing," CHEMOSPHERE Vol. 18 (1989), pg. 235.

Descriptor terms: dioxin; vernon houk; mo; syntex; times beach; pulp and paper industry; wildlife; cancer; immune system; birth defects; birds; fish; endocrine disruptors; health; epa; dioxin reassessment;

Friday, 28 March 2014

#250: Pet Dogs Get Cancer from Weed Killers (2,4-D)

=======================Electronic Edition========================

RACHEL'S HAZARDOUS WASTE NEWS #250
---September 11, 1991---
News and resources for environmental justice.
------ 
Environmental Research Foundation
P.O. Box 5036, Annapolis, MD 21403
Fax (410) 263-8944; Internet: erf@igc.apc.org
==========
RACHEL-4CM = DIOXIN FOCUSED DIRECTORY
Remote Access Chemical Hazards Electronic Library.
Dioxinnz.com
=======================Original Source========================

Pet dogs exposed to the weed killer 2,4-D are dying of cancer at twice normal rates, according to a study just published in the JOURNAL OF THE NATIONAL CANCER INSTITUTE.[1] Dog owners who spray or dust their lawns with weed killers containing 2,4-D are doubling Fido's chances of getting cancer, the study shows. Dogs walk across, or roll in, herbicide-treated lawns and then ingest toxic chemicals when they lick their coats or paws. Popular lawn-care products containing 2,4-D include Weedone, Weed-B-Gone, and many others (see below).[2]

Naturally, children who play on treated lawns will also come in contact with the chemical; dogs and children can also track the chemical indoors where prolonged exposure to humans may occur, but, so far as we know, no one has yet studied effects of weed killers on children or other family members inhabiting treated home sites.

In the past decade, several studies of farmers, and a few of railroad workers, have shown a connection between exposure to 2,4-D and an increased risk of human cancers. This latest study shows that dogs get some of the same kinds of cancers from weed killers that farmers in Nebraska,[3] Kansas,[4] and Saskatchewan,[5] and workers in Sweden[6,7,8] are reported to get from using 2,4-D on crops and to clear weeds along railroad tracks. In humans, the cancers are known as soft tissue sarcomas (STS), malignant lymphomas, and non-Hodgkin's lymphomas. In dogs, malignant lymphomas and non-Hodgkin's lymphomas predominate. The occurrence of such cancers in the American people has been rising slowly but steadily for several decades, tracking the emergence of the modern life style in which the dandelion-free lawn has come to symbolize the good life.[9] Non-Hodgkin's lymphomas--the kind dogs are reported to get most often from exposure to 2,4-D--have been the second fastest-growing cancer in humans in the U.S. during the past 15 years.

It is not clear exactly which components of weed killers are responsible for the cancers. There are three possible sources of the problem: (a) the active ingredient in the weed killer, (b) the so-called "inert" ingredients that are used as carriers for the chemicals that actually kill weeds, and (c) the dioxins that contaminate the active ingredients during manufacture.

About 600 million pounds of 2,4-D are spread on American soil each year by homeowners and farmers--about 60 million pounds of "active ingredients" and about 540 million pounds of "inert" ingredients that can include carbon tetrachloride (a carcinogen), chloroform (a carcinogen), chloroethane (a carcinogen) and 20 or more other ingredients that are labeled "inert" but which have well-known toxic properties. [10] Federal pesticide law does not require chemical companies to disclose what is in the "inert" ingredients in their products. Furthermore, federal law provides a $10,000 penalty for any government employee who reveals the make-up of "inert" ingredients in pesticides.

2,4-D and its closely-related chemical cousin, 2,4,5-T (which is now banned in the U.S.), are contaminated with dioxins during manufacture. Dioxins are extremely potent toxins that have a wide spectrum of effects in humans, wildlife, and laboratory animals (ses RHWN #249). A recent study by the National Institutes of Occupational Safety and Health (NIOSH) revealed a 46% cancer increase among workers in factories manufacturing these weed killers. (See RHWN #219.) Previous studies by scientists employed by herbicide manufacturing companies (BASF and Monsanto) had purported to show no effects in workers but there is now a growing concern among government officials that some of these studies were falsified. [11]

People spread 2,4-D around their homes and gardens to kill broad-leaf weeds, crab grass and dandelions. Farmers use it on tomatoes to cause all fruits to ripen at the same time for machine harvesting, and to increase the red color in potatoes. Utility companies, highway departments, and railroads use it to clear brush beneath power transmission lines and along highways and tracks. It is used heavily on corn, sorghum, rice and other crops to keep weeds down. From 1962 to 1971 during the Vietnam War, 2,4-D and its chemical cousin 2,4,5-T mixed together formed Agent Orange; it was sprayed by soldiers and airmen to defoliate the jungle where the Vietcong were living. Thousands of GIs have filed lawsuits against the U.S. government and against individual companies that supplied components of Agent Orange, such as Monsanto, Dow, Uniroyal, Hercules, Diamond Shamrock and others. A recent study in the AMERICAN JOURNAL OF PUBLIC HEALTH reveals that Vietnam veterans are 70% more likely to father children with one or more major birth defects, compared to men with no military service; it is unclear whether herbicide exposure is the most important cause. [12]

Common names for weed killers containing 2,4-D include Weedone, Weed-B-Gon, Green Cross Weed No More 80, Lawn-Keep, Salvo, Red Devil Dry Weed Killer, De-Pester Ded-Weed, Plantgard, Dormon, Dormone, Brush Killer 64, Weed-Rhap, Bladex-B, Butoxy-D, Dicofur, Ipaner, Moxon, Netagrone, Pielik, U 46 DP, Verton 38, B-Selektonon, Silvaprop, Agricorn D, Acme LV 4, Acme LV 6, Coprider, D50, DMA 4, Emulsamine, Fernesta, Ferxone, Macondray, Pennamine, Tributon, Weedatul, Agroxone, Spritz-Hormin, Desormone, Decamine, Weedar, R-H Weed Rhap 20, and Scott's 4-XD Weed Control.[2]

--Peter Montague, Ph.D.

===============

[1] Howard M. Hayes and others, "Case-Control Study of Canine Malignant Lymphoma: Positive Association With Dog Owner's Use of 2,4-Dichlorophenoxyacetic Acid Herbicides," JOURNAL OF THE NATIONAL CANCER INSTITUTE Vol. 83 (Sept. 4, 1991), pgs. 1226-1231.

[2] Product names were gathered from a search on "2,4-D" in the National Library of Medicine's online Hazardous Substances Data Bank; to learn details of this online system, write National Library of Medicine, 8600 Rockville Pike, Bethesda, MD 20894.

[3] Shelia Hoar Zahm and others, "A Case-Control Study of Non-Hodgkin's Lymphoma and the Herbicide 2,4-Dichlorophenoxyacetic Acid (2,4-D) in Eastern Nebraska," EPIDEMIOLOGY Vol. 1 (September, 1990), pgs. 349-256.

[4] Shelia K. Hoar and others, "Agricultural Herbicide Use and Risk of Lymphoma and Soft-Tissue Sarcoma," JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION Vol. 256 (Sept. 5, 1986), pgs. 1141-1147; see RHWN #003b.

[5] A. Blair, "Herbicides and non-Hodgkin's lymphoma: New evidence from a study of Saskatchewan farmers," JOURNAL OF THE NATIONAL CANCER INSTITUTE Vol. 82 (1990), pgs. 544-545.

[6] Olav Axelson and others, "Herbicide Exposure and Tumor Mortality," SCANDINAVIAN JOURNAL OF WORK, ENVIRONMENT, AND HEALTH Vol. 6 (March, 1980), pgs. 73-79.

[7] Mikael Eriksson and others, "Exposure to Dioxins as a Risk Factor for Soft Tissue Sarcoma: A Population-Based Case-Control Study," JOURNAL OF THE NATIONAL CANCER INSTITUTE Vol. 82 (March 21, 199), pgs. 486-490.

[8] Lennart Hardell and Mikael Eriksson, "The Association Between Soft Tissue Sarcomas and Exposure to Phenoxyacetic Acids," CANCER Vol. 62 (Aug. 1, 1988), pgs. 652-656.

[9] Kenneth P. Cantor and Others, "Distribution of Non-Hodgkin's Lymphomas in the United States Between 1950 and 1975," CANCER RESEARCH Vol. 40 (August, 1980), pgs. 2645-2652.

[10] U.S. Environmental Protection Agency staff members estimate that use of 2,4-D "active ingredient" is between 57 and 62 million pounds per year. Federal law does not allow EPA officials to gather exact data. See PESTICIDE INDUSTRY SALES AND USAGE; 1987 MARKET ESTIMATES (Washington, DC: Economic Analysis Branch, Biological and Economic Analysis Division, Office of Pesticide Programs, [U.S.] Environmental Protection Agency, September 1988), Table 9. However "active ingredients" account for only about 10% of an herbicide like 2,4-D; see Susan Jaffe, Michael Surgan, and Timothy P. Urban, THE SECRET HAZARDS OF PESTICIDES: INERT INGREDIENTS (Albany, NY: Office of the Attorney General, June, 1991), Table 1. Free copies of this Attorney General's report are available through the mail by contacting Office of Public Information, NY State Department of Law, 120 Broadway, NY, NY 10271. You can try to place a phone order by calling (212) 341-2000.

[11] See, for example, Leslie Roberts, "Monsanto Studies Under Fire," SCIENCE Vol. 251 (February 8, 1991), pg. 626. A Monsanto public relations spokesperson says the company's studies of worker health were sound; nevertheless, Roberts reports, U.S. Environmental Protection Agency has recently opened a criminal investigation to determine whether Monsanto "falsified" three epidemiological studies of its workers. For further evidence of concern expressed by government officials, see RHWN #171.

[12] Ann Aschengrau and Richard R. Monson, "Paternal Military Service in Vietnam and the Risk of Late Adverse Pregnancy Outcomes," AMERICAN JOURNAL OF PUBLIC HEALTH Vol. 80 (October, 1990), pgs. 1218-1224.

Descriptor terms: 2,4-d; cancer; dogs; inert ingredients; herbicides; pesticides; farming; occupational safety and health; studies;